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Medical condition
CD25 deficiency or interleukin 2 receptor alpha deficiency is an immunodeficiency disorder associated with mutations in the interleukin 2 receptor alpha
CD25_deficiency
Lymphocyte receptor specific for Interleukin-2
growth factors being studied by endocrinologists and biochemists". CD25 deficiency PDB: 2B5IWang X, Rickert M, Garcia KC (November 2005). "Structure
IL-2_receptor
II deficiency (with mutations in CIITA, RFXANK, RFX5, or RFXAP) CD25 deficiency CD27 deficiency STAT5b deficiency ITK deficiency SH2D1A deficiency (XLP1)
List of primary immunodeficiencies
List_of_primary_immunodeficiencies
Medical condition
differential diagnosis sees that the following should be considered: CD25 deficiency STAT5B deficiency Severe combined immunodeficiency X linked thrombocytopenia
Autoimmune polyendocrine syndrome
Autoimmune_polyendocrine_syndrome
Medical condition
mechanisms of autoimmune enteropathy isn't well known but dysfunction or deficiency of CD25+CD4+ regulatory T cells may play a role. Numerous other illnesses
Autoimmune_enteropathy
Resulting from inborn deficiencies in immune system
normally. To be considered a primary immunodeficiency (PID), the immune deficiency must be inborn, not caused by secondary factors such as other disease
Primary_immunodeficiency
White blood cells of the immune system
FOXP3, and CD25 and are thought to be derived from the same lineage as naïve CD4+ cells. Because effector T cells also express CD4 and CD25, Treg cells
Regulatory_T_cell
Type of white blood cell
successfully pair a pre-TCR forms, and the cell downregulates CD25 and is termed a DN4 cell (CD25−CD44−). These cells then undergo a round of proliferation
T_cell
Overview of and topical guide to immunology
immunological disorders (autoimmune diseases, hypersensitivities, immune deficiency, transplant rejection); the physical, chemical and physiological characteristics
Outline_of_immunology
Medical condition
defect in Treg production. Those individuals do not have circulating CD4+/CD25+/FOXP3+ Treg cells. Reduced expression of FOXP3 has been described, and these
IPEX_syndrome
Protein-coding gene in the species Homo sapiens
"Expression of the integrin alpha Ebeta 7 identifies unique subsets of CD25+ as well as CD25- regulatory T cells". Proceedings of the National Academy of Sciences
Integrin_alpha_E
Immune response protein
cells (a/iTregs), also identified by other less specific markers such as CD25 or CD45RB. In animal studies, Tregs that express FOXP3 are critical in the
FOXP3
Immune response to yeast
yeast tolerance. This alteration is marked by increased activation markers, CD25/CD69, upon proliferative stimulation of T-helper lymphocytes.[citation needed]
Anti–Saccharomyces cerevisiae antibody
Anti–Saccharomyces_cerevisiae_antibody
Found in the epithelial layer of mucosal linings
CD103, activation marker CD69, granzyme B and perforin cytolytic granules. CD25 expression is lower in comparison with effector memory T cells. Expression
Intraepithelial_lymphocyte
Elimination of autoreactive lymphocytes by the immune system
distinguished by the expression of surface markers CD44 and CD25, with the DN1 cells being CD44+ CD25-. Similarly to the TSPs, the DN1 cells are still capable
Central_tolerance
Anti-inflammatory cytokine
extent, lymphocytes, namely type-II T helper cells (TH2), mast cells, CD4+CD25+Foxp3+ regulatory T cells, and in a certain subset of activated T cells and
Interleukin_10
Neuroscientist
Michal (2002-11-26). "Neuroprotective autoimmunity: naturally occurring CD4+CD25+ regulatory T cells suppress the ability to withstand injury to the central
Jonathan_Kipnis
Protein-coding gene in the species Homo sapiens
novel member of phospholipase A2 family, is associated with lymphotoxin deficiency". Genes Immun. 1 (3): 191–9. doi:10.1038/sj.gene.6363659. PMID 11196711
PLA2G2D
(August 2005). "Hassall's corpuscles instruct dendritic cells to induce CD4+CD25+ regulatory T cells in human thymus". Nature. 436 (7054): 1181–1185. doi:10
Thymic_mimetic_cells
State of unresponsiveness of the immune system
cells develop from Foxp3- CD25+ CD4+ cells while iTreg cells develop from Foxp3+ CD25- CD4- cells (both become Foxp3+ CD25+CD4+). nTreg cells, when activated
Immune_tolerance
Cell surface receptor found in humans
(PMNs or PMLs), B cells (B1a, MZ B, B2), and T cells, including Tregs (CD4+CD25+ regulatory T cells). In some cases, it occurs in a heterodimer (combination
Toll-like_receptor_2
Medical condition with rash and bleeding risk
J, Yu Y, Bussel JB, Yazdanbakhsh K (August 2008). "Defective circulating CD25 regulatory T cells in patients with chronic immune thrombocytopenic purpura"
Immune thrombocytopenic purpura
Immune_thrombocytopenic_purpura
Group of innate immune cells that are derived from common lymphoid progenitors
specific organ sites. In humans, ILC2s express CRTH2, KLRG1, SST2, CD161, and CD25. In mice, ILC2s express CD44, but not CD161. ILC2s require IL-7 for their
Innate_lymphoid_cell
Medication used as chemotherapy and to suppress the immune system
immunotherapy. Suggested mechanisms include: Elimination of T regulatory cells (CD4+CD25+ T cells) in naive and tumor-bearing hosts Induction of T cell growth factors
Cyclophosphamide
; et al. (2002). "Neuroprotective autoimmunity: naturally occurring CD4+CD25+ regulatory T cells suppress the ability to withstand injury to the central
Protective_autoimmunity
Form of diabetes mellitus
IL2RA variation lowers IL-2 signaling and contributes to diminished CD4+CD25+ regulatory T cell function". Journal of Immunology. 188 (9): 4644–4653.
Type_1_diabetes
Transcription factor and coding gene in humans
(MSMD). In knock-out mice prepared in the 90s, a low amount of CD4+ and CD25+ regulatory T-cells and almost no IFNa, b and g response was discovered,
STAT1
Protein-coding gene in humans
has been shown to be regulator of T-cell activation. BHLHE41 upregulates CD25 expression through a Stat6-dependent mechanism, which enhances the IL-2 receptor-mediated
BHLHE41
Type of immune cell
with anti-CD40 antibody and CpG bacterial DNA. They were also positive for CD25, CD71 and PD-L1 after stimulation by CpG bacterial DNA and through TLR9.
Regulatory_B_cell
Group of cytokines
interleukins, and rare deficiencies of a number of them have been described, all featuring autoimmune diseases or immune deficiency. The majority of interleukins
Interleukin
Medical condition
identifiers is the absence in expression of the surface antigens CD10, CD11c, CD25, CD103 and cyclin D1 – an important regulator of cell-cycle progression.
B-cell prolymphocytic leukemia
B-cell_prolymphocytic_leukemia
Immune cell found in connective tissue
kinase 2 (TK2) domain. This mutation, as well as expression of either CD2 or CD25 (confirmed by immunostaining or flow cytometry), are characteristic of primary
Mast_cell
Type of cell surface receptor
active. This effect then spreads to native T cells which then switch to a CD4+CD25+GITR+FoxP3+ T regulatory phenotype. Currently, the two most prominent areas
Co-receptor
Type of immune cell
Activated T cells also produce the alpha sub-unit of the IL-2 receptor (CD25 or IL-2R), enabling a fully functional receptor that can bind with IL-2,
T_helper_cell
(GPI) link to the cell surface. Is also known as Heat Stable Antigen (HSA). CD25 a type I transmembrane protein present on activated T cells, activated B
List of human clusters of differentiation
List_of_human_clusters_of_differentiation
InterPro Family
to attack and kill malignant (cancer-causing) tumor cells. Type II IFN deficiency has been linked to several types of cancer, including B-cell lymphoma
Interferon_gamma
Mammalian gene encoding V-ATPase enzyme
"Characterization of IL-10-secreting T cells derived from regulatory CD4+CD25+ cells by the TIRC7 surface marker". Journal of Immunology. 180 (9): 6054–6063
TCIRG1
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CD25 DEFICIENCY
CD25 DEFICIENCY
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CD25 DEFICIENCY
CD25 DEFICIENCY
CD25 DEFICIENCY
CD25 DEFICIENCY
CD25 DEFICIENCY
CD25 DEFICIENCY
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